Mouse reduced in osteosclerosis transporter functions as an organic anion transporter 3 and is localized at abluminal membrane of blood-brain barrier.

Article Details

Citation

Ohtsuki S, Kikkawa T, Mori S, Hori S, Takanaga H, Otagiri M, Terasaki T

Mouse reduced in osteosclerosis transporter functions as an organic anion transporter 3 and is localized at abluminal membrane of blood-brain barrier.

J Pharmacol Exp Ther. 2004 Jun;309(3):1273-81. Epub 2004 Feb 4.

PubMed ID
14762099 [ View in PubMed
]
Abstract

The "reduced in osteosclerosis" transporter (Roct), which shows decreased expression in the osteosclerosis (oc) mutant mouse, has high homology with rat and human organic anion transporter 3 (OAT3). However, its transport properties and involvement in bone turnover are poorly understood. Here, we examined Roct-mediated transport using a Xenopus laevis oocyte expression system. Roct-expressing oocytes exhibited uptake of [(3)H]estrone sulfate, [(3)H]p-aminohippuric acid, [(3)H]benzylpenicillin, [(3)H]estradiol 17beta-glucronide, [(3)H]indoxyl sulfate, [(14)C]indomethacin, [(3)H]homovanillic acid, [(3)H]cimetidine, [(14)C]glutarate, [(14)C]salicylic acid, and [(3)H]methotrexate. Furthermore, the uptake of [(3)H]benzylpenicillin by Roct coexpressed with Na(+)-dicarboxylate cotransporter was trans-stimulated by glutarate preloading, and [(3)H]estrone sulfate uptake showed a similar tendency, suggesting that Roct is a dicarboxylate exchanger. [(3)H]Benzylpenicillin uptake by Roct was inhibited by OAT3 substrates and inhibitors, and by sulfate or glucuronide conjugates, and compounds involved in bone turnover. Roct mRNA is expressed abundantly in the kidney and was also detected in the brain, choroid plexus, and eye. Immunohistochemical analysis revealed that Roct is localized in brain capillary endothelial cells. These results indicate that the transport properties and tissue distribution of Roct are similar to those of OAT3, suggesting that Roct functions as mouse OAT3. Because Roct is expressed in the kidney and at the blood-brain barrier, it may play a role in the excretion of substrates such as conjugates and bone turnover factors.

DrugBank Data that Cites this Article

Drug Transporters
DrugTransporterKindOrganismPharmacological ActionActions
Aminohippuric acidSolute carrier family 22 member 8ProteinHumans
Unknown
Substrate
Inhibitor
Details
BenzylpenicillinSolute carrier family 22 member 8ProteinHumans
Unknown
Substrate
Inhibitor
Details
CimetidineSolute carrier family 22 member 8ProteinHumans
Unknown
Substrate
Inhibitor
Details
Conjugated estrogensSolute carrier family 22 member 8ProteinHumans
Unknown
Substrate
Inhibitor
Details
DinoprostoneSolute carrier family 22 member 8ProteinHumans
Unknown
Substrate
Inhibitor
Details
Glutaric AcidSolute carrier family 22 member 8ProteinHumans
Unknown
Substrate
Inhibitor
Details
IndomethacinSolute carrier family 22 member 8ProteinHumans
Unknown
Inhibitor
Details
LiothyronineSolute carrier family 22 member 8ProteinHumans
Unknown
Inhibitor
Details
LiotrixSolute carrier family 22 member 8ProteinHumans
Unknown
Inhibitor
Details
MethotrexateSolute carrier family 22 member 8ProteinHumans
Unknown
Substrate
Inhibitor
Details
PravastatinSolute carrier family 22 member 8ProteinHumans
No
Substrate
Inhibitor
Details
ProbenecidSolute carrier family 22 member 8ProteinHumans
Unknown
Inhibitor
Details
Salicylic acidSolute carrier family 22 member 8ProteinHumans
Unknown
Inhibitor
Details
Taurocholic acidSolute carrier family 22 member 8ProteinHumans
Unknown
Substrate
Inhibitor
Details